CTICasesBudd-Chiari Syndrome with Systemic-to-Pulmonary Collaterals
Imaging findings
CT abdomen and lower chest show signs of chronic liver disease, ascites, and massive superficial chest wall and paraspinal systemic collateral veins. These large veins are seen penetrating the pleural space and directly communicating with the pulmonary veins, leading to systemic-to-pulmonary vascular shunting.
Key takeaways
Budd-Chiari syndrome (hepatic vein occlusion) leads to portal hypertension and the development of extensive systemic collateral pathways. Systemic-to-pulmonary venous communications (most commonly between internal mammary or chest wall veins and pulmonary veins) are rare, but can occur, mimicking pulmonary arteriovenous malformations and leading to right-to-left shunting and hypoxia.
AI-assisted summary — may contain errors. Verify against the source video. Learn more
More from this webinar
- Incarcerated Diaphragmatic Gastric Hernia post Liver TransplantIatrogenic
- Tracheal Injury from Gunshot WoundTrauma
- Bentall Graft Infection with DehiscenceInfection
- Buerger's Disease with Renal InfarctionVascular
- Lap-Band Induced Pseudo-AchalasiaIatrogenic
- Postoperative Seroma in Hernia SacIatrogenic
- Infective Endocarditis with Massive Mitral VegetationInfection
- Portopulmonary ShuntVascular
See all cases from May 16, 2020 →
Related Vascular cases
- Septic Lakes in Interstitial Pulmonary Edema
- Intramural Blood Pools in Aortic Intramural Hematoma
- Hyperdense Pulmonary Embolism on Non-Contrast CT
- Stanford Type A Aortic Dissection
- Secondary Pulmonary Hemosiderosis from Mitral Heart Disease
- Mosaic Perfusion in Chronic Thromboembolic Pulmonary Hypertension